Site Map    
Dr. Erin Elster    
Upper Cervical Healthcare
 
Skip Navigation Links

Multiple Sclerosis


Description

The pathological process involved in Multiple Sclerosis(MS), a demyelinating disease, is the loss of myelin sheaths surrounding axons in the central nervous system (brain and spinal cord). Common early manifestations of Multiple Sclerosis (MS] include paresthesias (numbness / tingling in extremities), optic neuritis (vision loss), mild sensory or motor symptoms in a limb, and cerebellar incoordination (balance loss).

Although the most common course of Multiple Sclerosis (MS) is a relapsing and remitting pattern over many years, the manifestation in each patient varies. In most cases, as the disease progresses, remissions become less complete. Some patients have only a few brief episodes of disability, whereas others have a relentless downhill course over months or weeks. Although not all patients become disabled, the end stage of MS often can include ataxia (inability to coordinate voluntary movement), incontinence, paraplegia, and mental dysfunction due to widespread cerebral and spinal cord demyelination.



How Upper Cervical Care Relates to Multiple Sclerosis

While medical science has not determined the exact cause of MS, recent research is pointing towards a possible trauma-induced origin for MS.1-12 Evidence supports that trauma (in particular mild concussive injury to the head, neck or upper back) increases the risk of MS onset and/or formation of MS lesions.1-12 Following the trauma, MS symptoms can take months or years to develop.

The purpose of upper cervical care is to reverse the trauma-induced upper neck injury; thereby eliminating adverse effects upon the brain. While many MS sufferers recall specific traumas such as head injuries, auto accidents or falls, some do not. An evaluation is necessary in each individual's case to assess whether an upper cervical injury is present and whether benefit from upper cervical care can be achieved.

 

 

Research Summary


By 2019, approximately 75 MS patients have been examined and treated by Dr. Elster using specific Upper Cervical Care (see publications below). All patients showed evidence of trauma-induced upper cervical injuries during examination (upper cervical radiographs and digital infrared imaging). Many patients recalled specific incidences of trauma (auto accidents, falls, concussions, whiplashes) that could have caused their upper cervical injuries while some did not. Some traumas had occurred more than 10 years prior to the onset of MS symptoms. Even if a patient did not recall experiencing an accident or trauma to his or her neck, upper cervical injuries were found in 100% of cases examined.



Case Studies

View Case Studies related to Multiple Sclerosis


Research Articles and Publications
References:
  1. Chaudhuri A, Behan PO. Acute cervical hyperextension-hyperflexion injury may precipitate and/or exacerbate symptomatic multiple sclerosis. Eur J Neurol. 2001 Nov; 8(6): 109-10.
  2. Christie B. Multiple sclerosis linked with trauma in court case. BMJ (BMJ) 1996 Nov 16; 313 (7067): 1228.
  3. Christie B. Appeal overturns link between multiple sclerosis and whiplash. BMJ (BMJ) 1998 Mar; 316: 797.
  4. Poser CM. Trauma to the central nervous system may result in formation or enlargement of multiple sclerosis plaques. Arch Neurol 2000 Jul; 57(5): 1074-7.
  5. Poser CM. The role of trauma in the pathogenesis of multiple sclerosis: a review. Clin Neurol Neurosurg 1994 May; 96(2): 103-10. Poser CM. The role of trauma in the pathogenesis of multiple sclerosis: a review. Clin Neurol Neurosurg 1994 May; 96(2): 103-10. 6. Poser CM. The pathogenesis of multiple sclerosis. Additional considerations. J Neurol Sci 1993 Apr; 115 Suppl: S3-15.
  6. Martinelli V. Trauma, stress, and multiple sclerosis. Neurol Sci 2000; 21(4 Suppl 2): S849-52.
  7. Casetta I, Granieri E. Prognosis of multiple sclerosis: environmental factors. Neurol Sci 2000; 21 (4 Suppl 2): S839-42.
  8. Rudez J, Antonelli L, Materljan E. Injuries in the etiopathogenesis of multiple sclerosis. Lijec Vjesn 1998 Jan-Feb; 120(1-2): 24-7.
  9. Gusev E, Boiko A, Lauer K. Environmental risk factors in MS: a case-control study in Moscow. Acta Neurol Scand 1996 Dec; 94(6): 386-94.
  10. Morrison W, Nelson J. Environmental triggers in Multiple Sclerosis. Fact of fallacy? Axone 1994 Sep; 16(1): 23-6.
  11. Traynelis VC, Hitchon PW, Yuh WT. Magnetic resonance imaging and posttraumatic Lhermitte's Sign. J Spinal Disord 1990 Dec; 3(4): 376-9.
  12. Goetz CG, Pappert EJ. Trauma and movement disorders. Neurol Clin (NEU) 1992 Nov; (4): 907-19.

The content and materials provided in this web site are for informational and educational purposes only and are not intended to supplement or comprise a medical diagnosis or other professional opinion, or to be used in lieu of a consultation with a physician or competent health care professional for medical diagnosis and/or treatment. All content and materials including research papers, case studies and testimonials summarizing patients' responses to care are intended for educational purposes only and do not imply a guarantee of benefit. Individual results may vary, depending upon several factors including age of the patient, severity of the condition, severity of the spinal injury, and duration of time the condition has been present.